Inability to sustain intraphagolysosomal killing of Staphylococcus aureus predisposes to bacterial persistence in macrophages-Reference-Cited by-同舟云学术

Inability to sustain intraphagolysosomal killing of Staphylococcus aureus predisposes to bacterial persistence in macrophages

Published:2015-09-02 Issue:1 Volume:18 Page:80-96
ISSN:1462-5814
Container-title:Cellular Microbiology
language:en
Short-container-title:Cellular Microbiology

Author:

Jubrail Jamil¹²,Morris Paul¹²,Bewley Martin A.¹²,Stoneham Simon¹²,Johnston Simon A.¹²³,Foster Simon J.²³,Peden Andrew A.⁴,Read Robert C.⁵,Marriott Helen M.¹²,Dockrell David H.¹²⁶

Affiliation:

1. Department of Infection and Immunity University of Sheffield Sheffield UK

2. The Florey Institute University of Sheffield Sheffield UK

3. Department of Molecular Biology and Biotechnology University of Sheffield Sheffield UK

4. Department of Biomedical Sciences University of Sheffield Sheffield UK

5. Academic Unit of Clinical and Experimental Sciences University of Southampton Medical School Southampton UK

6. Academic Directorate of Communicable Diseases Sheffield Teaching Hospitals Sheffield UK

Publisher

Hindawi Limited

Subject

Virology,Immunology,Microbiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/cmi.12485

Reference73 articles.

1. Alveolar macrophages in pulmonary host defence – the unrecognized role of apoptosis as a mechanism of intracellular bacterial killing

2. SLAM is a microbial sensor that regulates bacterial phagosome functions in macrophages

3. A Cardinal Role for Cathepsin D in Co-Ordinating the Host-Mediated Apoptosis of Macrophages and Killing of Pneumococci

4. Proteomic Evaluation and Validation of Cathepsin D Regulated Proteins in Macrophages Exposed to Streptococcus pneumoniae

5. Pneumolysin Activates Macrophage Lysosomal Membrane Permeabilization and Executes Apoptosis by Distinct Mechanisms without Membrane Pore Formation

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