In Type 1 Diabetes Immunocompetent Cells are Defective in IL-16 Secretion
Author:
Publisher
Wiley
Subject
Immunology,General Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-3083.2011.02630.x/fullpdf
Reference5 articles.
1. Preferential migration of T regulatory cells induced by IL-16;McFadden;J Immunol,2007
2. Processing and activation of pro-interleukin-16 by caspase-3;Zhang;J Biol Chem,1998
3. Defective lymphocyte caspase-3 expression in type 1 diabetes mellitus;Vendrame;Eur J Endocrinol,2005
4. Impaired caspase-3 expression by peripheral T cells in chronic autoimmune thyroiditis and in autoimmune polyendocrine syndrome-2;Vendrame;J Clin Endocrinol Metab,2006
5. IL-21 drives secondary autoimmunity in patients with multiple sclerosis, following therapeutic lymphocyte depletion with alemtuzumab (Campath-1H);Jones;J Clin Invest,2009
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1. IFN-γ, SCF, MIP1b and IL-16 Were Associated with Risk of Diabetic Nephropathy: A Mendelian Randomization Study;Diabetes, Metabolic Syndrome and Obesity;2024-02
2. IL16 and IL18 gene polymorphisms in women with gestational diabetes;Ginekologia Polska;2017-05-31
3. Advances in the cellular immunological pathogenesis of type 1 diabetes;Journal of Cellular and Molecular Medicine;2014-03-14
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