Suppression of Experimental Autoimmune Myasthenia Gravis After CD8 Depletion is Associated with Decreased IFN-gamma and IL-4
Author:
Publisher
Wiley
Subject
Immunology,General Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-3083.1995.tb03680.x/fullpdf
Reference24 articles.
1. Myasthenia Gravis
2. Myasthenia Gravis
3. Ultrastructural Localization of the Terminal and Lytic Ninth Complement Component (C9) at the Motor End-plate in Myasthenia Gravis
4. Induction of Interferon-γ, Interleukin-4, and Transforming Growth Factor-β in Rats Orally Tolerized against Experimental Autoimmune Myasthenia Gravis
5. Regulation of Antibody Production by Helper T Cell Clones in Experimental Autoimmune Myasthenia Gravis Is Mediated by IL-4 and Antigen-Specific T Cell Factors
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2. Anti-CD8 Monoclonal Antibody Therapy Is Effective in the Prevention and Treatment of Experimental Autoimmune Glomerulonephritis;Journal of the American Society of Nephrology;2002-02
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4. Normalization of elevated CD4?/CD8? (double-negative) T cells after thymectomy parallels clinical remission in myasthenia gravis associated with thymic hyperplasia but not thymoma;Annals of Neurology;2000-10
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