No Evidence for TCR Vbeta Repertoire Changes Influencing Disease Protection in E-Transgenic NOD Mice
Author:
Publisher
Wiley
Subject
Immunology,General Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-3083.1995.tb03636.x/fullpdf
Reference32 articles.
1. The Murine Autoimmune Diabetes Model: NOD and Related Strains
2. The NOD Mouse: Recessive Diabetogenic Gene in the Major Histocompatibility Complex
3. MHC-Linked Protection from Diabetes Dissociated from Clonal Deletion of T Cells
4. The first external domain of the nonobese diabetic mouse class II I-A beta chain is unique.
5. Prevention of autoimmune insulitis by expression of I–E molecules in NOD mice
Cited by 4 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Differential MHC expression requirements for positive selection of separate TCR Vb families;Immunogenetics;1999-01-01
2. MHC class II Ab diabetogenic residue 57 Asp/non-Asp dimorphism influences T-cell recognition and selection;Immunogenetics;1998-01-22
3. Major Histocompatibility Complex–linked Control of Autoimmunity;Journal of Experimental Medicine;1997-10-06
4. Alleviation of insulitis in NOD mice is associated with expression of transgenic MHC E molecules on primary antigen‐presenting cells;Immunology;1997-04
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