Mechanisms of innate and acquired resistance to Cryptosporidium parvum infection in SCID mice
Author:
Publisher
Wiley
Subject
Immunology,Parasitology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1365-3024.1994.tb00354.x/fullpdf
Reference27 articles.
1. Susceptibility of major histocompatibility complex (MHC) class I- and MHC class II-deficient mice to Cryptosporidium parvum infection
2. Natural Immunity: A T-Cell-Independent Pathway of Macrophage Activation, Defined in the scid Mouse
3. A severe combined immunodeficiency mutation in the mouse
4. Requirement of endogenous interferon-gamma production for resolution of Listeria monocytogenes infection.
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1. Intestinal cDC1s provide cues required for CD4+ T cell–mediated resistance to Cryptosporidium;Journal of Experimental Medicine;2024-06-03
2. Dendritic cell-mediated responses to secreted Cryptosporidium effectors promote parasite-specific CD8+ T cell responses;Mucosal Immunology;2024-06
3. Intestinal cDC1s provide IL-12 dependent and independent functions required for CD4+T cell-mediated resistance toCryptosporidium;2023-11-13
4. Dendritic cell-mediated responses to secretedCryptosporidiumeffectors are required for parasite-specific CD8+T cell responses;2023-08-18
5. Early immune and host cell responses to Cryptosporidium infection;Frontiers in Parasitology;2023-01-27
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