Extracellular Domain of Pemphigus Vulgaris Antigen (Desmoglein 3) Mediates Weak Homophilic Adhesion
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Dermatology,Molecular Biology,Biochemistry
Reference24 articles.
1. Pemphigus: skin failure mediated by autoantibodies;Stanley;JAMA,1990
2. Autoantibodies against a novel epithelial cadherin in pemphigus vulgaris, a disease of cell adhesion;Amagai;Cell,1991
3. Cadherins: a molecular family important in selective cell-cell adhesion;Takeichi;Anna Rev Biochem,1990
4. Structure and interactions of desmosomal and other cadherins;Buxton;Semin Cell Biol,1992
5. Complete amino acid sequence of the epidermal desmoglein precursor polypeptide and identification of a second type of desmoglein gene;Koch;Eur J Cell Biol,1991
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1. Atomic Force Microscopy Provides New Mechanistic Insights into the Pathogenesis of Pemphigus;Frontiers in Immunology;2018-03-28
2. Desmoglein-3;Encyclopedia of Signaling Molecules;2018
3. Multiplexed autoantigen microarrays identify HLA as a key driver of anti-desmoglein and -non-desmoglein reactivities in pemphigus;Proceedings of the National Academy of Sciences;2016-02-01
4. Desmoglein-3;Encyclopedia of Signaling Molecules;2016
5. Bullous Diseases: Old Blisters with New Therapeutic Targets;Personalized Treatment Options in Dermatology;2015
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