Knockout of LASP1 in CXCR4 expressing CML cells promotes cell persistence, proliferation and TKI resistance-Reference-Cited by-同舟云学术

Knockout of LASP1 in CXCR4 expressing CML cells promotes cell persistence, proliferation and TKI resistance

Published:2020-01-19 Issue:5 Volume:24 Page:2942-2955
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J Cell Mol Med

Author:

Herrmann Andreas B.¹^ORCID,Müller Martha‐Lena²,Orth Martin F.³,Müller Jörg P.⁴,Zernecke Alma¹,Hochhaus Andreas⁵,Ernst Thomas⁵,Butt Elke¹,Frietsch Jochen J.⁵^ORCID

Affiliation:

1. Institut für Experimentelle Biomedizin II Universitätsklinikum Würzburg Würzburg Germany

2. Rudolf‐Virchow‐Zentrum für Experimentelle Biomedizin I Universitätsklinikum Würzburg Würzburg Germany

3. Labor für Pädiatrische Sarkombiologie Medizinische Fakultät Pathologisches Institut LMU München München Germany

4. Center for Molecular Biomedicine Institut für Molekulare Zellbiologie Universitätsklinikum Jena Jena Germany

5. Abteilung für Hämatologie und internistische Onkologie Klinik für Innere Medizin II Universitätsklinikum Jena Jena Germany

Funder

Deutsche Krebshilfe

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.14910

Reference45 articles.

1. Minimal Residual Disease Eradication in CML: Does It Really Matter?

2. Long-Term Outcomes of Imatinib Treatment for Chronic Myeloid Leukemia

3. Impact of comorbidities on overall survival in patients with chronic myeloid leukemia: results of the randomized CML Study IV

4. Treatment-free remission in patients with chronic myeloid leukemia

5. Discontinuation of imatinib in patients with chronic myeloid leukaemia who have maintained complete molecular remission for at least 2 years: the prospective, multicentre Stop Imatinib (STIM) trial

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