Hyperbaric oxygen activates visfatin expression and angiogenesis via angiotensin II and JNK pathway in hypoxic human coronary artery endothelial cells-Reference-Cited by-同舟云学术

Hyperbaric oxygen activates visfatin expression and angiogenesis via angiotensin II and JNK pathway in hypoxic human coronary artery endothelial cells

Published:2020-01-19 Issue:4 Volume:24 Page:2434-2443
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J Cell Mol Med

Author:

Chiu Chiung‐Zuan¹²^ORCID,Wang Bao‐Wei¹³,Yu Ying‐Ju³,Shyu Kou‐Gi²

Affiliation:

1. School of Medicine College of Medicine Fu Jen Catholic University New Taipei Taiwan

2. Division of Cardiology Shin‐Kong Wu Ho‐Su Memorial Hospital Taipei Taiwan

3. Central Laboratory Shin‐Kong Wu Ho‐Su Memorial Hospital Taipei Taiwan

Funder

Shin Kong Wu Ho-Su Memorial Hospital

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.14926

Reference37 articles.

1. The metabolic syndrome and adipocytokines

2. Therapy Insight: adipocytokines in metabolic syndrome and related cardiovascular disease

3. Visfatin: A Protein Secreted by Visceral Fat That Mimics the Effects of Insulin

4. Visfatin in adipocytes is upregulated by hypoxia through HIF1α-dependent mechanism

5. Hypoxic induction of human visfatin gene is directly mediated by hypoxia-inducible factor-1

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