Kir6.1 improves cardiac dysfunction in diabetic cardiomyopathy via the AKT‐FoxO1 signalling pathway-Reference-Cited by-同舟云学术

Kir6.1 improves cardiac dysfunction in diabetic cardiomyopathy via the AKT‐FoxO1 signalling pathway

Published:2021-02-06 Issue:8 Volume:25 Page:3935-3949
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J Cell Mol Med

Author:

Wang Jinxin¹²^ORCID,Bai Jing¹,Duan Peng³,Wang Hao²,Li Yang¹,Zhu Qinglei¹

Affiliation:

1. Department of Cardiology Chinese PLA General Hospital Beijing China

2. Department of Geriatric Cardiology Chinese PLA General HospitalNational Clinical Research Center for Geriatric Diseases Beijing China

3. Department of Cardiology Chinese PLA No. 371 Hospital Henan China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.16346

Reference55 articles.

1. Molecular mechanisms of diabetic cardiomyopathy

2. Of mice and men: models and mechanisms of diabetic cardiomyopathy

3. Activation of Foxo1 by Insulin Resistance Promotes Cardiac Dysfunction and β–Myosin Heavy Chain Gene Expression

4. Myocardial Loss of IRS1 and IRS2 Causes Heart Failure and Is Controlled by p38α MAPK During Insulin Resistance

5. Opening of mitoKATP improves cardiac function and inhibits apoptosis via the AKT‐Foxo1 signaling pathway in diabetic cardiomyopathy;Duan P;Int J Mol Med,2018

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