The Ca2+channel subunit CaVβ2a‐subunit down‐regulates voltage‐activated ion current densities by disrupting actin‐dependent traffic in chromaffin cells
Author:
Affiliation:
1. Facultad de Ciencias, Centro Interdisciplinario de Neurociencia de Valparaíso, Instituto de Neurociencia Universidad de Valparaíso Valparaíso Chile
Funder
Fondo Nacional de Desarrollo Científico y Tecnológico
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience,Biochemistry
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/jnc.14851
Reference60 articles.
1. Calcium channel subtypes differentially regulate fusion pore stability and expansion
2. IP3 Receptor-Dependent Cytoplasmic Ca2+ Signals Are Tightly Controlled by Cavβ3
3. “Actin”g on GLUT4: Membrane & Cytoskeletal Components of Insulin Action
4. The β Subunit of Voltage-Gated Ca2+Channels
5. Coexpression of Cloned α1B, β2a, and α2/δ Subunits Produces Non-Inactivating Calcium Currents Similar to Those Found in Bovine Chromaffin Cells
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1. Exocytosis, endocytosis and recycling of secretory vesicles in neuroendocrine cells, and its regulation by cortical actin;BIOCELL;2022
2. Actin dynamics as critical ion channel regulator: ENaC and Piezo in focus;American Journal of Physiology-Cell Physiology;2021-05-01
3. Voltage‐gated calcium channel nanodomains: molecular composition and function;The FEBS Journal;2021-02-25
4. The interplay between α7 nicotinic acetylcholine receptors, pannexin‐1 channels and P2X7 receptors elicit exocytosis in chromaffin cells;Journal of Neurochemistry;2020-09-30
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