miR‐499 released during myocardial infarction causes endothelial injury by targeting α7‐nAchR-Reference-Cited by-同舟云学术

miR‐499 released during myocardial infarction causes endothelial injury by targeting α7‐nAchR

Published:2019-07-03 Issue:9 Volume:23 Page:6085-6097
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J Cell Mol Med

Author:

Zhou Rui¹,Huang Wenjun¹,Fan Xinrong²,Liu Feng³,Luo Liangqin³,Yuan Haiyang¹,Jiang Yu¹,Xiao Haiying¹,Zhou Zhichao⁴,Deng Chenliang⁵,Dang Xitong¹³^ORCID

Affiliation:

1. The Key Laboratory of Medical Electrophysiology of Ministry of Education and Medical Electrophysiological Key Laboratory of Sichuan Province, Collaborative Innovation Center for Prevention and Treatment of Cardiovascular Disease of Sichuan Province, Institute of Cardiovascular Research Southwest Medical University Stockholm Sweden

2. Department of Cardiovascular Medicine The 1st Affiliated Hospital of Southwest Medical University, Southwest Medical University Stockholm Sweden

3. Department of Cardiovascular Surgery The 1st Affiliated Hospital of Southwest Medical University, Southwest Medical University Stockholm Sweden

4. Division of Cardiology, Department of Medicine, Heart and Vascular Theme Karolinska University Hospital, Karolinska Institute Stockholm Sweden

5. Department of Plastic Surgery Shanghai 6^th People’s Hospital, Shanghai Jiaotong University School of Medicine Shanghai China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.14474

Reference39 articles.

1. Cardiac Endothelial-Myocardial Signaling: Its Role in Cardiac Growth, Contractile Performance, and Rhythmicity

2. Systemic Atherosclerotic Inflammation Following Acute Myocardial Infarction: Myocardial Infarction Begets Myocardial Infarction

3. RNAi targeting multiple cell adhesion molecules reduces immune cell recruitment and vascular inflammation after myocardial infarction

4. Myocardial infarction accelerates atherosclerosis

5. Targeting Interleukin-1β Reduces Leukocyte Production After Acute Myocardial Infarction

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