Small junction, big problems: Neuromuscular junction pathology in mouse models of amyotrophic lateral sclerosis (ALS)-Reference-Cited by-同舟云学术

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Small junction, big problems: Neuromuscular junction pathology in mouse models of amyotrophic lateral sclerosis (ALS)

Published:2021-06-07 Issue: Volume: Page:
ISSN:0021-8782
Container-title:Journal of Anatomy
language:en
Short-container-title:J Anat

Author:

Alhindi Abrar¹²³,Boehm Ines¹³^ORCID,Chaytow Helena¹³^ORCID

Affiliation:

1. Edinburgh Medical School Biomedical Sciences University of Edinburgh Edinburgh UK

2. Department of Anatomy Faculty of Medicine King Abdulaziz University Jeddah Saudi Arabia

3. Euan MacDonald Centre for Motor Neurone Disease Research University of Edinburgh Edinburgh UK

Funder

Anatomical Society

King Abdulaziz University

Publisher

Wiley

Subject

Cell Biology,Developmental Biology,Molecular Biology,Ecology, Evolution, Behavior and Systematics,Histology,Anatomy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/joa.13463

Reference120 articles.

1. Terminal Schwann cells at the human neuromuscular junction;Alhindi A.;Brain Communications,2021

2. Effect of locally delivered IGF‐1 on nerve regeneration during aging: an experimental study in rats;Apel P.J.;Muscle and Nerve,2010

3. Early and persistent abnormal decoding by glial cells at the neuromuscular junction in an ALS model;Arbour D.;Journal of Neuroscience,2015

4. ALS‐linked TDP‐43 mutations produce aberrant RNA splicing and adult‐onset motor neuron disease without aggregation or loss of nuclear TDP‐43;Arnold E.S.;Proceedings of the National Academy of Sciences of the United States of America,2013

5. Complement activation at the motor end‐plates in amyotrophic lateral sclerosis;Bahia El Idrissi N.;Journal of Neuroinflammation,2016

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