Smad3: A Key Player in Pathogenetic Mechanisms Dependent on TGF-β-Reference-Cited by-同舟云学术

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Smad3: A Key Player in Pathogenetic Mechanisms Dependent on TGF-β

Published:2003-05 Issue:1 Volume:995 Page:1-10
ISSN:0077-8923
Container-title:Annals of the New York Academy of Sciences
language:en
Short-container-title:

Author:

ROBERTS ANITA B.,RUSSO ANGELO,FELICI ANGELINA,FLANDERS KATHLEEN C.

Publisher

Wiley

Subject

History and Philosophy of Science,General Biochemistry, Genetics and Molecular Biology,General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1749-6632.2003.tb03205.x/fullpdf

Reference44 articles.

1. 1Roberts, A.B. & M.B. Sporn. 1990. The transforming growth factors-β. In Handbook of Experimental Pharmacology: Peptide Growth Factors and Their Receptors, pp. 419-472. Springer-Verlag. New York/Berlin.

2. The TGF-beta superfamily: new members, new receptors, and new genetic tests of function in different organisms;Kingsley;Genes Dev.,1994

3. Expression cloning of the TGF-beta type II receptor, a functional transmembrane serine/threonine kinase;Lin;Cell,1992

4. TGF-beta receptor signaling;Derynck;Biochim. Biophys. Acta,1997

5. TGF-beta signaling: positive and negative effects on tumorigenesis;Wakefield;Curr. Opin. Genet. Dev.,2002

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