Familial British Dementia: Expression and Metabolism of BRI-Reference-Cited by-同舟云学术

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Familial British Dementia: Expression and Metabolism of BRI

Published:2006-01-25 Issue:1 Volume:920 Page:93-99
ISSN:0077-8923
Container-title:Annals of the New York Academy of Sciences
language:en
Short-container-title:

Author:

KIM SEONG-HUN,WANG RONG,GORDON DAVID J.,BASS JOSEPH,STEINER DONALD F.,THINAKARAN GOPAL,LYNN DAVID G.,MEREDITH STEPHEN C.,SISODIA SANGRAM S.

Publisher

Wiley

Subject

History and Philosophy of Science,General Biochemistry, Genetics and Molecular Biology,General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1749-6632.2000.tb06909.x/fullpdf

Reference17 articles.

1. Familial cerebral amyloid angiopathy with nonneuritic amyloid plaque formation;Plant;Brain,1990

2. A form of familial presenile dementia with spastic paralysis (including the pathological examination of a case);Worster-Drought;Brain,1940

3. Congophilic angiopathy of the brain: a clinical and pathological report on two siblings;Griffiths;J. Neurol. Neurosurg. Psychiatry,1982

4. A stop-codon mutation in the BRI gene associated with familial British dementia;Vidal;Nature,1999

5. Furin: a mammalian subtilisin/Kex2p-like endoprotease involved in processing of a wide variety of precursor proteins;Nakayama;Biochem. J.,1997

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