Safety and Efficacy of Quinidine Sulfate in Slow-Channel Congenital Myasthenic Syndromea
Author:
Publisher
Wiley
Subject
History and Philosophy of Science,General Biochemistry, Genetics and Molecular Biology,General Neuroscience
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1749-6632.1998.tb10929.x/fullpdf
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1. A newly recognized congenital myasthenic syndrome attributed to a prolonged open time of the acetylcholine-induced ion channel
2. Mutation of the acetylcholine receptor α subunit causes a slow-channel myasthenic syndrome by enhancing agonist binding affinity
3. New mutations in acetylcholine receptor subunit genes reveal heterogeneity in the slow-channel congenital myasthenic syndrome
4. Effects of the quinoline derivatives quinine, quinidine, and chloroquine on neuromuscular transmission
5. Variables influencing neuropathic endpoints: The Rochester Diabetic Neuropathy Study of Healthy Subjects
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4. Mutations Causing Slow-Channel Myasthenia Reveal That a Valine Ring in the Channel Pore of Muscle AChR is Optimized for Stabilizing Channel Gating;Human Mutation;2016-08-21
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