Antibodies Affecting Ion Channel Function in Acquired Neuromyotonia, in Seropositive and Seronegative Myasthenia Gravis, and in Antibody-mediated Arthrogryposis Multiplex Congenita-Reference-Cited by-同舟云学术

Antibodies Affecting Ion Channel Function in Acquired Neuromyotonia, in Seropositive and Seronegative Myasthenia Gravis, and in Antibody-mediated Arthrogryposis Multiplex Congenita

Published:1998-05 Issue:1 MYASTHENIA GR Volume:841 Page:482-496
ISSN:0077-8923
Container-title:Annals of the New York Academy of Sciences
language:en
Short-container-title:Annals NY Acad Sci

Author:

VINCENT ANGELA,JACOBSON LESLIE,PLESTED PAUL,POLIZZI AGATA,TANG TERESA,RIEMERSMA SIETSKE,NEWLAND CLAIRE,GHORAZIAN SARA,FARRAR JEREMY,MACLENAN CAL,WILLCOX NICHOLAS,BEESON DAVID,NEWSDOM-DAVIS JOHN

Publisher

Wiley

Subject

History and Philosophy of Science,General Biochemistry, Genetics and Molecular Biology,General Neuroscience

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1749-6632.1998.tb10968.x/fullpdf

Reference38 articles.

1. Immunological associations of acquired neuromyotonia (Isaacs' syndrome)

2. Autoimmune aetiology for acquired neuromyotonia (Isaacs' syndrome)

3. Autoimmune aetiology for acquired neuromyotonia

4. 4Wintzen, A. R., J. G. Van Dijk & A. BRAND. 1994. Neuromyotonia with early response to plasmapheresis associated with proximal action myoclonus with late response to plasmapheresis. Muscle Nerve 17(suppl. 1): S221.

5. Acquired neuromyotonia: Evidence for autoantibodies directed against K+ channels of peripheral nerves

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