Functional Relevance of an Enhanced Expression of the Na+-Ca2+ Exchanger in the Failing Human Heart
Author:
Publisher
Wiley
Subject
History and Philosophy of Science,General Biochemistry, Genetics and Molecular Biology,General Neuroscience
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1749-6632.1996.tb44832.x/fullpdf
Reference10 articles.
1. Active Na+ and Ca2+ transport, Na+-Ca2+ exchange, and intracellular Na+ and Ca2+ content in young spontaneously hypertensive rats;David-Dufilho;J. Cardiovasc. Pharmacol.,1986
2. Sarcolemmal Ca2+ transport activities in cardiac hypertrophy caused by pressure overload;Nakanishi;Am. J. Physiol.,1989
3. Gene expression of the cardiac Na+-Ca2+ exchanger in end-stage human heart failure;Studer;Circ. Res.,1994
4. Evidence for a sustained effectiveness of sodium-channel activator in failing human myocardium;Schwinger;J. Mol. Cell. Cardiol,1991
5. Molecular cloning and functional expression of the cardiac sarcolemmal Na+-Ca2+ exchanger;Nicoll;Science,1990
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3. Cellular and molecular determinants of altered Ca2+handling in the failing rabbit heart: primary defects in SR Ca2+uptake and release mechanisms;American Journal of Physiology-Heart and Circulatory Physiology;2007-03
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