Adrenal necrosis in a dog receiving trilostane for the treatment of hyperadrenocorticism

Author:

Chapman P. S.1,Kelly D. F.2,Archer J.3,Brockman D. J.4,Neiger R.5

Affiliation:

1. Department of Clinical Studies, School of Veterinary Medicine, University of Pennsylvania, 3900 Delancey Street, Philadelphia, PA 19104, USA

2. Department of Veterinary Pathology, University of Liverpool, Liverpool L69 3BX

3. Department of Pathology and Infectious Diseases, Royal Veterinary College, University of London, Hawkshead Lane, North Mymms, Hatfield, Hertfordshire AL9 7TA

4. Department of Veterinary Clinical Sciences, Royal Veterinary College, University of London, Hawkshead Lane, North Mymms, Hatfield, Hertfordshire AL9 7TA

5. Klinik für Klientiere, Justus‐Liebig Universität Giessen, Franfurterstrasse 126, D‐35392 Giessen, Germany

Publisher

Wiley

Subject

Small Animals

Reference24 articles.

1. Results of non-selective adrenocorticolysis by O,p'-DDD in 129 dogs with pituitary-dependent hyperadrenocorticism

2. THE HUMAN ADRENAL GLAND WITH SPECIAL REFERENCE TO THE VASCULATURE

3. Eastwood J. M.&Elwood C. M.(2003)Prolonged hypoadrenocorticism in five dogs treated with trilostane for pituitary dependent hyperadrenocorticism. Proceedings of the British Small Animal Veterinary Association. April. p587.

4. Venous infarction of the adrenal glands

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