Nucleotide substitutions in CD101 , the human homolog of a diabetes susceptibility gene in non-obese diabetic mouse, in patients with type 1 diabetes-Reference-Cited by-同舟云学术

Nucleotide substitutions in CD101 , the human homolog of a diabetes susceptibility gene in non-obese diabetic mouse, in patients with type 1 diabetes

Published:2016-11-25 Issue:3 Volume:8 Page:286-294
ISSN:2040-1116
Container-title:Journal of Diabetes Investigation
language:en
Short-container-title:J Diabetes Investig

Author:

Okuno Misako¹²³,Kasahara Yoshihito³⁴,Onodera Masafumi⁵,Takubo Noriyuki³⁶,Okajima Michiko³⁴,Suga Shigeru³⁷,Watanabe Nobuyuki⁵,Suzuki Junichi¹²³,Ayabe Tadayuki¹³,Urakami Tatsuhiko²³,Kawamura Tomoyuki³⁸,Kikuchi Nobuyuki³⁹,Yokota Ichiro³¹⁰,Kikuchi Toru³¹¹,Amemiya Shin³¹¹,Nakabayashi Kazuhiko¹²,Hayashi Keiko¹²,Hata Kenichiro¹²,Matsubara Yoichi¹³,Ogata Tsutomu¹³¹⁴,Fukami Maki¹³,Sugihara Shigetaka³¹⁵

Affiliation:

1. Department of Molecular Endocrinology; National Research Institute for Child Health and Development; Tokyo Japan

2. Department of Pediatrics; Nihon University School of Medicine; Tokyo Japan

3. The Japanese Study Group of Insulin Therapy for Childhood and Adolescent Diabetes; Kanazawa Japan

4. Department of Pediatrics; Kanazawa University; Kanazawa Japan

5. Department of Human Genetics; National Research Institute for Child Health and Development; Tokyo Japan

6. Department of Pediatrics; Juntendo University; Tokyo Japan

7. Department of Pediatrics; National Hospital Organization Mie Hospital; Tsu Japan

8. Department of Pediatrics; Osaka City University; Osaka Japan

9. Department of Pediatrics; Yokohama City Minato Red Cross Hospital; Yokohama Japan

10. Department of Pediatrics; Division of Pediatric Endocrinology and Metabolism; Shikoku Medical Center for Children and Adults; Zentsuji Japan

11. Department of Pediatrics; Saitama Medical University; Saitama Japan

12. Department of Maternal-Fetal Biology; National Research Institute for Child Health and Development; Tokyo Japan

13. Department of Institute Director; National Research Institute for Child Health and Development; Tokyo Japan

14. Department of Pediatrics; Hamamatsu University School of Medicine; Hamamatsu Japan

15. Department of Pediatrics; Tokyo Women's Medical University Medical Center East; Tokyo Japan

Funder

National Center Biobank Network

Japan Society for the Promotion of Science

Ministry of Health, Labour and Welfare

Japan Agency for Medical Research and Development

National Center for Child Health and Development

Takeda Foundation

Japan Diabetes Foundation

Manpei Suzuki Diabetes Foundation

Publisher

Wiley

Subject

General Medicine,Endocrinology, Diabetes and Metabolism,Internal Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jdi.12586/fullpdf

Reference24 articles.

1. Immunogenetics of type 1 diabetes mellitus;Morran;Mol Aspects Med,2015

2. Candidate genes for type 1 diabetes modulate pancreatic islet inflammation and β-cell apoptosis;Santin;Diabetes Obes Metab,2013

3. Type I diabetes mellitus: genetic factors and presumptive enteroviral etiology or protection;Precechtelova;J Pathog,2014

4. Genetics of diabetes-are we missing the genes or the disease?;Groop;Mol Cell Endocrinol,2014

5. Finding the missing heritability of complex diseases;Manolio;Nature,2009

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4. CD101 genetic variants modify regulatory and conventional T cell phenotypes and functions;Cell Reports Medicine;2021-06

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