Oligodendroglial deletion of ESCRT-I component TSG101 causes spongiform encephalopathy-Reference-Cited by-同舟云学术

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Oligodendroglial deletion of ESCRT-I component TSG101 causes spongiform encephalopathy

Published:2016-08-23 Issue:11 Volume:108 Page:324-337
ISSN:0248-4900
Container-title:Biology of the Cell
language:en
Short-container-title:Biol. Cell

Author:

Walker Will P.¹,Oehler Abby²,Edinger Aimee L.³,Wagner Kay-Uwe⁴,Gunn Teresa M.¹

Affiliation:

1. McLaughlin Research Institute; Great Falls MT 59405 USA

2. Department of Pathology; Institute for Neurodegenerative Diseases; University of California; San Francisco CA 94143 USA

3. Department of Developmental and Cell Biology; University of California; Irvine CA 92697 USA

4. Eppley Institute for Research in Cancer and Allied Diseases; University of Nebraska Medical Center; Omaha NE 68198 USA

Funder

The McLaughlin Research Institute

Publisher

Wiley

Subject

Cell Biology,General Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/boc.201600014/fullpdf

Reference64 articles.

1. Prion strains are differentially released through the exosomal pathway;Arellano-Anaya;Cell. Mol. Life. Sci.,2015

2. Spongiform encephalopathy in AIDS dementia complex: report of five cases;Artigas;J. Acquir. Immune. Defic. Syndr.,1989

3. The major myelin-resident protein PLP is transported to myelin membranes via a transcytotic mechanism: involvement of sulfatide;Baron;Mol. Cell. Biol.,2015

4. The pathogenesis of transmissible spongiform encephalopathy: an ultrastructural study;Beck;Brain,1982

5. Temporal and spatial organization of ESCRT protein recruitment during HIV-1 budding;Bleck;Proc. Natl. Acad. Sci. U.S.A.,2014

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