Trafficking and localisation to the plasma membrane of Nav1.5 promoted by the β2 subunit is defective due to a β2 mutation associated with Brugada syndrome-Reference-Cited by-同舟云学术

Trafficking and localisation to the plasma membrane of Nav1.5 promoted by the β2 subunit is defective due to a β2 mutation associated with Brugada syndrome

Published:2017-07 Issue:7 Volume:109 Page:273-291
ISSN:0248-4900
Container-title:Biology of the Cell
language:en
Short-container-title:Biol. Cell

Author:

Dulsat Gemma¹²,Palomeras Sonia¹²,Cortada Eric¹²,Riuró Helena¹²,Brugada Ramon¹²³,Vergés Marcel¹²³^ORCID

Affiliation:

1. Cardiovascular Genetics Group; Girona Biomedical Research Institute (IDIBGI); Salt Girona 17190 Spain

2. Biomedical Research Networking Center on Cardiovascular Diseases (CIBERCV); ISCIII Madrid 28029 Spain

3. Medical Sciences Department; University of Girona Medical School; Girona 17003 Spain

Funder

Spanish Ministerio de Sanidad y Consumo

“la Caixa” Foundation

Ministerio de Economía y Competitividad

Publisher

Wiley

Subject

Cell Biology,General Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/boc.201600085/fullpdf

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5. Scn2b deletion in mice results in ventricular and atrial arrhythmias;Bao;Circ. Arrhythm. Electrophysiol.,2016

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