The role of dysregulated PI3Kdelta signaling in human autoimmunity*-Reference-Cited by-同舟云学术

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The role of dysregulated PI3Kdelta signaling in human autoimmunity*

Published:2022-01-28 Issue:1 Volume:307 Page:134-144
ISSN:0105-2896
Container-title:Immunological Reviews
language:en
Short-container-title:Immunological Reviews

Author:

Bier Julia¹²,Deenick Elissa K.¹³^ORCID

Affiliation:

1. Garvan Institute of Medical Research Darlinghurst New South Wales Australia

2. St Vincent’s Clinical School Faculty of Medicine and Health UNSW Sydney Sydney New South Wales Australia

3. Faculty of Medicine and Health UNSW Sydney Sydney New South Wales Australia

Funder

American Association of Immunologists

National Health and Medical Research Council

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/imr.13067

Reference109 articles.

1. PI3K isoforms in cell signalling and vesicle trafficking

2. Signaling by the Phosphoinositide 3-Kinase Family in Immune Cells

3. The PI3K Pathway in Human Disease

4. p110δ, a Novel Phosphatidylinositol 3-Kinase Catalytic Subunit That Associates with p85 and Is Expressed Predominantly in Leukocytes

5. Signal transduction via CD40 involves activation of lyn kinase and phosphatidylinositol-3-kinase, and phosphorylation of phospholipase C gamma 2.

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1. Both T and B cells contribute to dysregulated activation and differentiation of CD4⁺T cells in Activated PI3K delta syndrome 1;2024-08-04

2. Discovery of Novel Azaindoles as Potent and Selective PI3Kδ Inhibitors for Treatment of Multiple Sclerosis;Journal of Medicinal Chemistry;2024-05-16

3. Up‐regulation of miR‐192‐5p inhibits the ELAVL1/PI3Kδ axis and attenuates microvascular endothelial cell proliferation, migration and angiogenesis in diabetic retinopathy;Diabetic Medicine;2023-03-27

4. Human PIK3R1 mutations disrupt lymphocyte differentiation to cause activated PI3Kδ syndrome 2;Journal of Experimental Medicine;2023-03-21

5. Oncogenic potential of PIK3CD in glioblastoma is exerted through cytoskeletal proteins PAK3 and PLEK2;Laboratory Investigation;2022-12

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