PU.1 inhibition attenuates atrial fibrosis and atrial fibrillation vulnerability induced by angiotensin‐II by reducing TGF‐β1/Smads pathway activation-Reference-Cited by-同舟云学术

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PU.1 inhibition attenuates atrial fibrosis and atrial fibrillation vulnerability induced by angiotensin‐II by reducing TGF‐β1/Smads pathway activation

Published:2021-06-15 Issue:14 Volume:25 Page:6746-6759
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J Cell Mol Med

Author:

Hu Juan¹,Zhang Jing‐Jing²,Li Li³,Wang Shan‐Ling¹,Yang Hai‐Tao¹,Fan Xian‐Wei¹,Zhang Lei‐Ming¹,Hu Guang‐Ling¹,Fu Hai‐Xia¹,Song Wei‐Feng¹,Yan Li‐Jie¹,Liu Jing‐Jing¹,Wu Jin‐Tao¹^ORCID,Kong Bin²

Affiliation:

1. Heart Center of Henan Provincial People’s Hospital Central China Fuwai HospitalCentral China Fuwai Hospital of Zhengzhou University Zhengzhou China

2. Department of Cardiology Renmin Hospital of Wuhan University Hubei China

3. Department of Cardiology Qitai Farm Hospital Xinjiang China

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.16678

Reference73 articles.

1. Cardiac Fibrosis in Patients With Atrial Fibrillation

2. Update on atrial fibrillation

3. Myeloperoxidase acts as a profibrotic mediator of atrial fibrillation

4. Evaluating the Atrial Myopathy Underlying Atrial Fibrillation

5. Paracrine Signals Modulate Atrial Epicardial Progenitor Cells and Development of Subepicardial Adiposity and Fibrosis Implications for Atrial Fibrillation

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2. M2a macrophages regulate fibrosis and affect the outcome after stroke via PU.1/mTOR pathway in fibroblasts;Neurochemistry International;2024-02

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