GPR126 regulates colorectal cancer cell proliferation by mediating HDAC2 and GLI2 expression-Reference-Cited by-同舟云学术

GPR126 regulates colorectal cancer cell proliferation by mediating HDAC2 and GLI2 expression

Published:2021-03-19 Issue:5 Volume:112 Page:1798-1810
ISSN:1347-9032
Container-title:Cancer Science
language:en
Short-container-title:Cancer Sci

Author:

Cui Hengxiang¹²^ORCID,Yu Wenjie³,Yu Minhao⁴,Luo Yang⁴,Yang Mingming⁵,Cong Ruochen¹,Chu Xin¹,Gao Ganglong⁴,Zhong Ming⁴

Affiliation:

1. Medical Research Center Second Affiliated Hospital of Nantong University Nantong China

2. Institute of Neuroscience State Key Laboratory of Neuroscience CAS Center for Excellence in Brain Science and Intelligence Technology Chinese Academy of Sciences Shanghai China

3. Department of Internal Medicine Carver College of MedicineThe University of Iowa Iowa City IA USA

4. Department of Gastrointestinal Surgery School of Medicine Renji HospitalShanghai Jiao Tong University Shanghai China

5. Department of Biochemistry and Molecular Biology School of Medicine Nantong University Nantong China

Funder

National Natural Science Foundation of China-Guangdong Joint Fund

Publisher

Wiley

Subject

Cancer Research,Oncology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/cas.14868

Reference47 articles.

1. A G Protein–Coupled Receptor Is Essential for Schwann Cells to Initiate Myelination

2. Gpr126/Adgrg6 Has Schwann Cell Autonomous and Nonautonomous Functions in Peripheral Nerve Injury and Repair

3. Analysis of Gpr126 function defines distinct mechanisms controlling the initiation and maturation of myelin

4. Mutations of GPR126 Are Responsible for Severe Arthrogryposis Multiplex Congenita

5. GPR126 Protein Regulates Developmental and Pathological Angiogenesis through Modulation of VEGFR2 Receptor Signaling

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