DEK‐targeting aptamer DTA‐64 attenuates bronchial EMT‐mediated airway remodelling by suppressing TGF‐β1/Smad, MAPK and PI3K signalling pathway in asthma-Reference-Cited by-同舟云学术

DEK‐targeting aptamer DTA‐64 attenuates bronchial EMT‐mediated airway remodelling by suppressing TGF‐β1/Smad, MAPK and PI3K signalling pathway in asthma

Published:2020-10-30 Issue:23 Volume:24 Page:13739-13750
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J. Cell. Mol. Med.

Author:

Song Yilan¹²³,Wang Zhiguang¹⁴,Jiang Jingzhi¹²,Piao Yihua¹⁵,Li Li¹²,Xu Chang¹²,Piao Hongmei¹⁴,Li Liangchang¹²,Yan Guanghai¹²^ORCID

Affiliation:

1. Jilin Key Laboratory for Immune and Targeting Research on Common Allergic Diseases Yanbian University Yanji China

2. Department of Anatomy, Histology and Embryology Yanbian University Medical College Yanji China

3. Postdoctoral Programme, Research Center Affiliated Hospital of Yanbian University Yanji China

4. Department of Respiratory Medicine Affiliated Hospital of Yanbian University Yanji China

5. Department of Intensive Care Unit Affiliated Hospital of Yanbian University Yanji China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.15942

Reference34 articles.

1. Asthma

2. Immune induction of airway remodeling

3. Epithelial mesenchymal transition (EMT): a universal process in lung diseases with implications for cystic fibrosis pathophysiology

4. Beyond epithelial-to-mesenchymal transition: Common suppression of differentiation programs underlies epithelial barrier dysfunction in mild, moderate, and severe asthma

5. DEK promoted EMT and angiogenesis through regulating PI3K/AKT/mTOR pathway in triple-negative breast cancer

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