AML1 protein interacts with influenza A virus neuraminidase and upregulates IFN‐β response in infected mammalian cells-Reference-Cited by-同舟云学术

AML1 protein interacts with influenza A virus neuraminidase and upregulates IFN‐β response in infected mammalian cells

Published:2020-02-17 Issue:4 Volume:70 Page:252-258
ISSN:0266-8254
Container-title:Letters in Applied Microbiology
language:en
Short-container-title:Lett Appl Microbiol

Author:

Gaur P.¹,Kumar P.²³,Sharma A.⁴,Lal S.K.²³⁵^ORCID

Affiliation:

1. School of Pharmacy Faculty of Medicine The Hebrew University of Jerusalem Jerusalem Israel

2. Department of Biotechnology Mewar University Chittorgarh Rajasthan India

3. Virology Group International Centre for Genetic Engineering & Biotechnology New Delhi India

4. Department of Microbiology and Molecular Genetics Faculty of Medicine The Institute for Medical Research – Israel‐Canada (IMRIC) The Hebrew University Jerusalem Israel

5. School of Science Monash University Malaysia Selangor DE Malaysia

Publisher

Wiley

Subject

Applied Microbiology and Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/lam.13279

Reference30 articles.

1. Core-binding factors in hematopoiesis and immune function

2. Virus Entry, Assembly, Budding, and Membrane Rafts

3. RUNX1–ETO induces a type I interferon response which negatively effects t(8;21)-induced increased self-renewal and leukemia development

4. Transcription Factor Runx3 Is Induced by Influenza A Virus and Double-Strand RNA and Mediates Airway Epithelial Cell Apoptosis

5. Influenza virus and cell signaling pathways

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. RUNX1 inhibits the antiviral immune response against influenza A virus through attenuating type I interferon signaling;Virology Journal;2022-03-05