Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats (一氧化氮与内皮素对糖尿病雌性大鼠主动脉血管内皮维持血管收缩反应的影响)

Author:

Sartoretto Simone M.1,Santos-Eichler Rosângela1,de Cássia A. Tostes Rita2,Carvalho Maria Helena C.1,Akamine Eliana H.1,Fortes Zuleica B.1

Affiliation:

1. Department of Pharmacology; Institute of Biomedical Sciences; University of São Paulo; São Paulo; Brazil

2. Department of Pharmacology; Medical School of Ribeirão Preto; University of São Paulo; Ribeirão Preto; Brazil

Publisher

Wiley

Subject

Endocrinology, Diabetes and Metabolism

Reference45 articles.

1. Inhibition of nitric oxide synthase by l-NAME improves ventricular performance in streptozotocin-diabetic rats;Smith;J Mol Cell Cardiol,1997

2. Inhibition of iNOS augments cardiovascular action of norepinephrine in streptozotocin-induced diabetes;Cheng;Cardiovasc Res,2004

3. Short-term streptozotocin-induced diabetes induces pressure decrease associated with reduced aortic 45Ca2+ uptake and selective depression of the sustained noradrenergic contraction;Rebolledo;Diabetes Metab,2001

4. Increased expression of iNOS is associated with endothelial dysfunction and impaired pressor responsiveness in streptozotocin-induced diabetes;Nagareddy;Am J Physiol Heart Circ Physiol,2005

5. Vascular reactivity in diabetes mellitus: Role of the endothelial cell;Fortes;Br J Pharmacol,1983

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