Spontaneous arising of a lymphoblastoid B‐cell line harbouring a pre‐leukemic DNMT3A mutation in acute myeloid leukaemia cell culture-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

Spontaneous arising of a lymphoblastoid B‐cell line harbouring a pre‐leukemic DNMT3A mutation in acute myeloid leukaemia cell culture

Published:2021-10-14 Issue:22 Volume:25 Page:10778-10782
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J Cell Mol Med

Author:

Hayun Michal¹^ORCID,Szwarcwort Moran²,Rosenberg Dina³,Sahar Dvora³,Ofran Yishai¹⁴⁵^ORCID

Affiliation:

1. The Clinical Research Institute at Rambam (CRIR) Rambam Health Care Campus Haifa Israel

2. Virology Laboratory Rambam Health Care Campus Haifa Israel

3. Hematology Laboratory Rambam Health Care Campus Haifa Israel

4. Department of Hematology and Bone Marrow Transplantation Rambam Health Care Campus Haifa Israel

5. Department of Hematology Shaare Zedek Medical Center Faculty of Medicinethe Hebrew University of Jerusalem Jerusalem Israel

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.16992

Reference15 articles.

1. Identification of pre-leukaemic haematopoietic stem cells in acute leukaemia

2. Polyclonal Ig production after Epstein-Barr virus infection of human lymphocytes in vitro

3. Epstein-Barr virus and human diseases: recent advances in diagnosis

4. Cyclosporin A promotes spontaneous outgrowth in vitro of Epstein–Barr virus-induced B-cell lines

5. Spontaneous production of epstein-barr virus by b lymphoblastoid cell lines obtained from patients with sjögren's syndrome. possible involvement of a novel strain of epstein-barr virus in disease pathogenesis

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP