Nicotine-induced molecular alterations are modulated by GABAB receptor activity-Reference-Cited by-同舟云学术

Nicotine-induced molecular alterations are modulated by GABAB receptor activity

Published:2017-04-17 Issue:1 Volume:23 Page:230-246
ISSN:1355-6215
Container-title:Addiction Biology
language:en
Short-container-title:Addiction Biology

Author:

Varani Andres P.¹,Pedrón Valeria T.¹,Aon Amira J.¹,Höcht Christian²,Acosta Gabriela B.¹,Bettler Bernhard³,Balerio Graciela N.¹²^ORCID

Affiliation:

1. CONICET - Universidad de Buenos Aires, Instituto de Investigaciones Farmacológicas (ININFA); Buenos Aires Argentina

2. Universidad de Buenos Aires, Facultad de Farmacia y Bioquímica; Cátedra de Farmacología; Buenos Aires Argentina

3. Department of Biomedicine, Institute of Physiology, Pharmazentrum; University of Basel; Switzerland

Funder

Consejo Nacional de Investigaciones Científicas y Técnicas

Universidad de Buenos Aires

Schweizerischer Nationalfonds zur Förderung der Wissenschaftlichen Forschung

Publisher

Wiley

Subject

Psychiatry and Mental health,Pharmacology,Medicine (miscellaneous)

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/adb.12506/fullpdf

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4. Nicotine-induced antinociception, rewarding effects, and physical dependence are decreased in mice lacking the preproenkephalin gene;Berrendero;J Neurosci,2005

5. The NK1 receptor is essential for the full expression of noxious inhibitory controls in the mouse;Bester;J Neurosci,2001

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