Molecular deficiency (ies) in MT1 melatonin signaling pathway underlies the melatonin-unresponsive phenotype in MDA-MB-231 human breast cancer cells-Reference-Cited by-同舟云学术

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Molecular deficiency (ies) in MT1 melatonin signaling pathway underlies the melatonin-unresponsive phenotype in MDA-MB-231 human breast cancer cells

Published:2014-01-18 Issue:3 Volume:56 Page:246-253
ISSN:0742-3098
Container-title:Journal of Pineal Research
language:en
Short-container-title:J. Pineal Res.

Author:

Mao Lulu¹²,Yuan Lin¹²,Xiang Shulin¹²,Zeringue Samantha B.³,Dauchy Robert T.¹²⁴,Blask David E.¹²⁴,Hauch Adam³,Hill Steven M.¹²

Affiliation:

1. Department of Structural and Cellular Biology; Tulane University School of Medicine; New Orleans LA USA

2. Tulane Cancer Center and Louisiana Cancer Research Consortium; New Orleans LA USA

3. Department of Surgery; Tulane University School of Medicine; New Orleans LA USA

4. Laboratory of Chrono-Neuroendocrine Oncology; Tulane University School of Medicine; New Orleans LA USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Endocrinology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jpi.12117/fullpdf

Reference36 articles.

1. Cellular mechanisms of melatonin action;Vanecek;Physiol Rev,1998

2. Melatonin, the circadian multioscillator system and health: the need for detailed analyses of peripheral melatonin signaling;Hardeland;J Pineal Res,2012

3. Melatonin and its analogs in insomnia and depression;Cardinali;J Pineal Res,2012

4. Molecular pathways involved in seasonal body weight and reproductive responses governed by melatonin;Barrett;J Pineal Res,2012

5. Cloning and characterization of a mammalian melatonin receptor that mediates reproductive and circadian responses;Reppert;Neuron,1994

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