Complement split product C4d deposition in placenta in systemic lupus erythematosus and pregnancy-induced hypertension
Author:
Affiliation:
1. Department of Diagnostic Pathology; Kyoto University Hospital; Kyoto; Japan
2. Department of Gynecology and Obstetrics; Kyoto University Hospital; Kyoto; Japan
Publisher
Wiley
Subject
General Medicine,Pathology and Forensic Medicine
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/pin.12041/fullpdf
Reference24 articles.
1. Pregnancy Outcome in Systemic Lupus Erythematosus (SLE) is Improving: Results from a Case Control Study and Literature Review;Yan Yuen;Open Rheumatol J,2008
2. Placental pathology in systemic lupus erythematosus: A prospective study;Magid;Am J Obstet Gynecol,1998
3. Maternal vasculopathy and histologic diagnosis of preeclampsia: Poor correlation of histologic changes and clinical manifestation;Zhang;Am J Obstet Gynecol,2006
4. Classical complement activation as a footprint for murine and human antiphospholipid antibody-induced fetal loss;Cohen;J Pathol,2011
5. Excessive complement activation is associated with placental injury in patients with antiphospholipid antibodies;Shamonki;Am J Obstet Gynecol,2007
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1. Placental lesions in systemic lupus erythematosus pregnancies associated with small for gestational age infants;Rheumatology;2024-08-28
2. Virtual crossmatching reveals upregulation of placental HLA-Class II in chronic histiocytic intervillositis;Scientific Reports;2024-08-12
3. Pregnancy as a susceptible state for thrombotic microangiopathies;Frontiers in Medicine;2024-02-27
4. Exploration of antibody-mediated immunity in chronic histiocytic intervillositis reveals no evidence of maternal anti-fetal rejection;2023-06-26
5. Placental damage in pregnancies with systemic lupus erythematosus: A narrative review;Frontiers in Immunology;2022-08-17
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