The Akt inhibitor, triciribine, ameliorates chronic hypoxia-induced vascular pruning and TGFβ-induced pulmonary fibrosis-Reference-Cited by-同舟云学术

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The Akt inhibitor, triciribine, ameliorates chronic hypoxia-induced vascular pruning and TGFβ-induced pulmonary fibrosis

Published:2015-07-06 Issue:16 Volume:172 Page:4173-4188
ISSN:0007-1188
Container-title:British Journal of Pharmacology
language:en
Short-container-title:Br J Pharmacol

Author:

Abdalla Maha¹²,Sabbineni Harika¹,Prakash Roshini¹,Ergul Adviye¹³,Fagan Susan C¹⁴,Somanath Payaningal R¹⁵⁶⁷

Affiliation:

1. Clinical and Experimental Therapeutics; College of Pharmacy; University of Georgia and Charlie Norwood VA Medical Center; Augusta GA USA

2. Department of Pharmaceutical Sciences; South College School of Pharmacy; Knoxville TN USA

3. Department of Physiology; Georgia Regents University; Augusta GA USA

4. Department of Neurology; Georgia Regents University; Augusta GA USA

5. Department of Medicine; Georgia Regents University; Augusta GA USA

6. Vascular Biology Center; Georgia Regents University; Augusta GA USA

7. Cancer Center; Georgia Regents University; Augusta GA USA

Funder

National Institutes of Health

University of Georgia College of Pharmacy

Wilson Pharmacy Foundation

American Heart Association

Publisher

Wiley

Subject

Pharmacology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/bph.13203/fullpdf

Reference56 articles.

1. Akt1 mediates α-smooth muscle actin expression and myofibroblast differentiation via myocardin and serum response factor;Abdalla;J Biol Chem,2013

2. The Concise Guide to PHARMACOLOGY 2013/14: Enzymes;Alexander;Br J Pharmacol,2013

3. Simple method of estimating severity of pulmonary fibrosis on a numerical scale;Ashcroft;J Clin Pathol,1988

4. Nadph oxidase 4 is expressed in pulmonary artery adventitia and contributes to hypertensive vascular remodeling;Barman;Arterioscler Thromb Vasc Biol,2014

5. Increased AKT S473 phosphorylation after mTORC1 inhibition is rictor dependent and does not predict tumor cell response to PI3K/mTOR inhibition;Breuleux;Mol Cancer Ther,2009

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