A molecular mechanism for how sigma factor AlgT and transcriptional regulator AmrZ inhibit twitching motility in Pseudomonas aeruginosa
Author:
Affiliation:
1. State Key Laboratory of Microbial Resources Institute of Microbiology, Chinese Academy of Sciences Beijing 100101 China
2. University of Chinese Academy of Sciences Beijing 100049 China
Funder
National Natural Science Foundation of China
National Basic Research Program of China
Publisher
Wiley
Subject
Ecology, Evolution, Behavior and Systematics,Microbiology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/1462-2920.14985
Reference69 articles.
1. Pseudomonas aeruginosa AlgZ, a ribbon-helix-helix DNA-binding protein, is essential for alginate synthesis and algD transcriptional activation
2. The Pseudomonas aeruginosa Ribbon-Helix-Helix DNA-Binding Protein AlgZ (AmrZ) Controls Twitching Motility and Biogenesis of Type IV Pili
3. The Sigma Factor AlgU Plays a Key Role in Formation of Robust Biofilms by Nonmucoid Pseudomonas aeruginosa
4. Pseudomonas aeruginosa AlgR Regulates Type IV Pilus Biosynthesis by Activating Transcription of the fimU-pilVWXY1Y2E Operon
5. Pseudomonas aeruginosauses a cyclic-di-GMP-regulated adhesin to reinforce the biofilm extracellular matrix
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