Systemic mitochondrial disruption is a key event in the toxicity of bacterial pore‐forming toxins to Caenorhabditis elegans-Reference-Cited by-同舟云学术

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Systemic mitochondrial disruption is a key event in the toxicity of bacterial pore‐forming toxins to Caenorhabditis elegans

Published:2021-03-30 Issue:9 Volume:23 Page:4896-4907
ISSN:1462-2912
Container-title:Environmental Microbiology
language:en
Short-container-title:Environ Microbiol

Author:

Shi Jianwei¹^ORCID,Zhang Fengjuan¹,Chen Ling¹,Bravo Alejandra²,Soberón Mario²,Sun Ming¹^ORCID

Affiliation:

1. State Key Laboratory of Agricultural Microbiology, College of Life Science and Technology Huazhong Agricultural University Wuhan China

2. Departamento de Microbiología Molecular, Instituto de Biotecnología Universidad Nacional Autónoma de México Cuernavaca Morelos Mexico

Funder

National Basic Research Program of China

National Natural Science Foundation of China

Publisher

Wiley

Subject

Ecology, Evolution, Behavior and Systematics,Microbiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/1462-2920.15376

Reference49 articles.

1. Resistance to Bacillus thuringiensis Toxin in Caenorhabditis elegans from Loss of Fucose

2. Activation of Type I and III Interferon Response by Mitochondrial and Peroxisomal MAVS and Inhibition by Hepatitis C Virus

3. Ubiquitin-Like Protein 5 Positively Regulates Chaperone Gene Expression in the Mitochondrial Unfolded Protein Response

4. Increased mitochondrial fusion allows the survival of older animals in diverse C. elegans longevity pathways

5. HLH-30/TFEB-mediated autophagy functions in a cell-autonomous manner for epithelium intrinsic cellular defense against bacterial pore-forming toxin in C. elegans

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3. C. elegans monitor energy status via the AMPK pathway to trigger innate immune responses against bacterial pathogens;Communications Biology;2022-06-30

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