Computational Modeling of a Putative Fetal Alcohol Syndrome Mechanism
Author:
Publisher
Wiley
Subject
Psychiatry and Mental health,Toxicology,Medicine (miscellaneous)
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/j.1530-0277.1995.tb01029.x/fullpdf
Reference42 articles.
1. RECOGNITION OF THE FETAL ALCOHOL SYNDROME IN EARLY INFANCY
2. Publication trends in fetal alcohol, tobacco and narcotic effects
3. A Hypothetical Mechanism for Fetal Alcohol Syndrome Involving Ethanol Inhibition of Retinoic Acid Synthesis at the Alcohol Dehydrogenase Step
4. Hypothesis: Prenatal Ethanol-Induced Birth Defects and Retinoic Acid
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1. Alcohol Dehydrogenase 2*3 Affects Alterations in Offspring Facial Morphology Associated With Maternal Ethanol Intake in Pregnancy;Alcoholism: Clinical & Experimental Research;2004-10
2. Fetal ADH2*3, Maternal Alcohol Consumption, and Fetal Growth;International Journal of Toxicology;2004-01
3. Molecular docking studies on interaction of diverse retinol structures with human alcohol dehydrogenases predict a broad role in retinoid ligand synthesis;Biochimica et Biophysica Acta (BBA) - Protein Structure and Molecular Enzymology;1999-07
4. Alcohol dehydrogenases inXenopus development: Conserved expression of ADH1 and ADH4 in epithelial retinoid target tissues;Developmental Dynamics;1998-11
5. ADH1 and ADH4 alcohol/retinol dehydrogenases in the developing adrenal blastema provide evidence for embryonic retinoid endocrine function;Developmental Dynamics;1998-09
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