Fibrinogen-like protein-2 causes deterioration in cardiac function in experimental autoimmune myocarditis rats through regulation of programmed death-1 and inflammatory cytokines-Reference-Cited by-同舟云学术

Fibrinogen-like protein-2 causes deterioration in cardiac function in experimental autoimmune myocarditis rats through regulation of programmed death-1 and inflammatory cytokines

Published:2017-10-16 Issue:2 Volume:153 Page:246-252
ISSN:0019-2805
Container-title:Immunology
language:en
Short-container-title:Immunology

Author:

Zheng Zhenzhong¹²^ORCID,Yu Yinghui¹²³,Potla Ratnakar⁴,Wu Yujing¹²,Wu Hao⁴

Affiliation:

1. Department of Cardiology; The First Affiliated Hospital of Nanchang University; Nanchang Jiangxi China

2. Jiangxi Hypertension Research Institute; Nanchang Jiangxi China

3. People's Hospital of Qing hua dao; Qing hua dao Hebei China

4. Vascular Biology Program; Boston Children's Hospital Department of Surgery; Harvard Medical School; Boston MA USA

Funder

National Natural Science Foundation of China

Technology Support project of Jiangxi province

AHA SDG grant

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/imm.12837/fullpdf

Reference21 articles.

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4. PD-1 upregulated on regulatory T cells during chronic virus infection enhances the suppression of CD8+ T cell immune response via the interaction with PD-L1 expressed on CD8+ T cells;Park;J Immunol,2015

5. Lichtman AH.PD-1 protects against inflammation and myocyte damage in T cell-mediated myocarditis;Tarrio;J Immunol,2012

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