Protein kinase D1 induced epithelial–mesenchymal transition and invasion in salivary adenoid cystic carcinoma via E‐cadherin/Snail regulation

Author:

Du Yue1,Lv Die1,Cui Bomiao1,Li Xiaoying1,Chen Hongli1,Kang Yingzhu1,Chen Qianming12,Feng Yun1,Zhang Ping1,Chen Jiao1ORCID,Zhou Xuedong1ORCID

Affiliation:

1. State Key Laboratory of Oral Diseases National Clinical Research Center for Oral Diseases West China Hospital of StomatologySichuan University Chengdu China

2. Key Laboratory of Oral Biomedical Research of Zhejiang Province Affiliated Stomatology Hospital Zhejiang University School of Stomatology Hangzhou China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

General Dentistry,Otorhinolaryngology

Reference50 articles.

1. Integrin-Linked Kinase, Snail and Multidrug Resistance Protein 1: Three concordant players in the progression of non-small cell lung cancer

2. Protein Kinase D1 Maintains the Epithelial Phenotype by Inducing a DNA-Bound, Inactive SNAI1 Transcriptional Repressor Complex

3. Temporal progression of metastasis in lung: Cell survival, dormancy, and location dependence of metastatic inefficiency;Cameron M. D.;Cancer Research,2000

4. SOD2 deregulation enhances migration, invasion and has poor prognosis in salivary adenoid cystic carcinoma

5. Protein kinase D1 regulates hypoxic metabolism through HIF‐1alpha and glycolytic enzymes incancer cells;Chen J.;Oncology Reports,2018

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