E2F1 in neurons is cleaved by calpain in an NMDA receptor-dependent manner in a model of HIV-induced neurotoxicity-Reference-Cited by-同舟云学术

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E2F1 in neurons is cleaved by calpain in an NMDA receptor-dependent manner in a model of HIV-induced neurotoxicity

Published:2014-11-10 Issue:6 Volume:132 Page:742-755
ISSN:0022-3042
Container-title:Journal of Neurochemistry
language:en
Short-container-title:J. Neurochem.

Author:

Zyskind Jacob W.¹,Wang Ying¹,Cho Giyong¹,Ting Jenhao H.¹,Kolson Dennis L.²,Lynch David R.³⁴,Jordan-Sciutto Kelly L.¹

Affiliation:

1. Department of Pathology; School of Dental Medicine; University of Pennsylvania; Philadelphia Pennsylvania USA

2. Department of Neurology; The Perelman School of Medicine; University of Pennsylvania; Philadelphia Pennsylvania USA

3. Division of Neurology; Children's Hospital of Philadelphia; Philadelphia Pennsylvania USA

4. Department of Neurology; University of Pennsylvania; Philadelphia Pennsylvania USA

Funder

NIH

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/jnc.12956/fullpdf

Reference66 articles.

1. NMDA-receptor activation induces calpain-mediated beta-catenin cleavages for triggering gene expression;Abe;Neuron,2007

2. Site-specific hyperphosphorylation of pRb in HIV-induced neurotoxicity;Akay;Mol. Cell. Neurosci.,2011

3. Proline-rich sequences that bind to Src homology 3 domains with individual specificities;Alexandropoulos;Proc. Natl Acad. Sci. USA,1995

4. Conditional disruption of calpain in the CNS alters dendrite morphology, impairs LTP, and promotes neuronal survival following injury;Amini;J. Neurosci.,2013

5. Src-mediated tyrosine phosphorylation of NR2 subunits of N-methyl-D-aspartate receptors protects from calpain-mediated truncation of their C-terminal domains;Bi;J. Biol. Chem.,2000

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4. Confound, Cause, or Cure: The Effect of Cannabinoids on HIV-Associated Neurological Sequelae;Viruses;2021-06-26

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