Neocortical GABA release at high intracellular sodium and low extracellular calcium: an anti-seizure mechanism

Author:

Rassner Michael P.1,Moser Andreas23,Follo Marie4,Joseph Kevin15,van Velthoven-Wurster Vera6,Feuerstein Thomas J.13

Affiliation:

1. Section of Clinical Neuropharmacology of the Department of Neurosurgery; University of Freiburg; Freiburg Germany

2. Neurochemical Research Group; Department of Neurology; University of Lübeck; Lübeck Germany

3. Freiburg Institute for Advanced Studies (FRIAS); University of Freiburg; Freiburg Germany

4. Department of Medicine I; Medical Center - University of Freiburg; Freiburg Germany

5. Section of Neuroelectronic Systems of the Department of Neurosurgery; University of Freiburg; Freiburg Germany

6. Department of Neurosurgery; ZU Brussels; Belgium

Funder

Freiburg Institute for Advanced Studies, Albert-Ludwigs-Universität Freiburg

Albert-Ludwigs-Universität Freiburg

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Biochemistry

Reference58 articles.

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2. Inhibition of the Na+-Ca++ exchanger enhances anoxia and glucopenia induced [3H]aspartate release in hippocampal slices;Amoroso;J. Pharmacol. Exp. Ther.,1993

3. Tiagabine: efficacy and safety in partial seizures - current status;Bauer;Neuropsychiatr. Dis. Treat.,2008

4. Na+, K+-ATPase: functions in the nervous system and the involvement in neurologic disease;Benarroch;Neurology,2011

5. Proteomic screening of glutamatergic mouse brain synaptosomes isolated by fluorescence activated sorting;Biesemann;EMBO J.,2014

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