Monoamine oxidase-A is a novel driver of stress-induced premature senescence through inhibition of parkin-mediated mitophagy-Reference-Cited by-同舟云学术

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Monoamine oxidase-A is a novel driver of stress-induced premature senescence through inhibition of parkin-mediated mitophagy

Published:2018-07-12 Issue:5 Volume:17 Page:e12811
ISSN:1474-9718
Container-title:Aging Cell
language:en
Short-container-title:Aging Cell

Author:

Manzella Nicola¹²,Santin Yohan¹,Maggiorani Damien¹,Martini Hélène¹,Douin-Echinard Victorine¹,Passos Joao F.³,Lezoualc'h Frank¹,Binda Claudia²,Parini Angelo¹,Mialet-Perez Jeanne¹^ORCID

Affiliation:

1. Institute of Metabolic and Cardiovascular Diseases (I2MC); Institut National de la Santé et de la Recherche Médicale (INSERM), Université de Toulouse; Toulouse France

2. Department of Biology and Biotechnology; University of Pavia; Pavia Italy

3. Ageing Research Laboratories; Newcastle University Institute for Ageing, Newcastle University; Newcastle upon Tyne UK

Funder

Agence Nationale de la Recherche

Fondazione Cariplo

Région Occitanie

Fondation pour la Recherche Médicale

Publisher

Wiley

Subject

Cell Biology,Ageing

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/acel.12811/fullpdf

Reference41 articles.

1. Naturally occurring p16(Ink4a)-positive cells shorten healthy lifespan;Baker;Nature,2016

2. Oxidative damage to mitochondrial DNA is inversely related to maximum life span in the heart and brain of mammals;Barja;The FASEB Journal,2000

3. Oxidative stress by monoamine oxidase mediates receptor-independent cardiomyocyte apoptosis by serotonin and postischemic myocardial injury;Bianchi;Circulation,2005

4. Cellular senescence: When bad things happen to good cells;Campisi;Nature Reviews Molecular Cell Biology,2007

5. Cellular senescence in aging and age-related disease: From mechanisms to therapy;Childs;Nature Medicine,2015

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