N‐glycosylated IgG in patients with kidney transplants increases calcium/calmodulin kinase IV in podocytes and causes injury
Author:
Affiliation:
1. Department of Medicine Beth Israel Deaconess Medical Center Harvard Medical School Boston Massachusetts USA
2. Department of Pathology Beth Israel Deaconess Medical Center Harvard Medical School Boston Massachusetts USA
Funder
National Institute of Arthritis and Musculoskeletal and Skin Diseases
Publisher
Wiley
Subject
Pharmacology (medical),Transplantation,Immunology and Allergy
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/ajt.16140
Reference41 articles.
1. The Value of Protocol Biopsies to Identify Patients With De Novo Donor-Specific Antibody at High Risk for Allograft Loss
2. Podocyte Depletion Causes Glomerulosclerosis: Diphtheria Toxin–Induced Podocyte Depletion in Rats Expressing Human Diphtheria Toxin Receptor Transgene
3. The Two Kidney to One Kidney Transition and Transplant Glomerulopathy: A Podocyte Perspective
4. Podocytes Are Nonhematopoietic Professional Antigen-Presenting Cells
5. Regulation of glycogen synthase kinase-3 beta (GSK-3β) by the Akt pathway in gliomas
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