Deficiency of p38α in macrophage ameliorates d -galactosamine/TNF-α-induced acute liver injury in mice-Reference-Cited by-同舟云学术

Deficiency of p38α in macrophage ameliorates d -galactosamine/TNF-α-induced acute liver injury in mice

Published:2017-11-14 Issue:24 Volume:284 Page:4200-4215
ISSN:1742-464X
Container-title:The FEBS Journal
language:en
Short-container-title:FEBS J

Author:

Liu Jiao¹,Zhang Shengjie¹,Cao Hongchao¹,Wang Hui¹,Sun Chao¹,Liu Shengnan¹,Yu Shuxian¹,Li Yan¹,Liu Wei¹,Wang Hui²,Jiang Jingjing³,Ying Hao¹⁴

Affiliation:

1. Key Laboratory of Food Safety Research; Institute for Nutritional Sciences; Shanghai Institutes for Biological Sciences; Chinese Academy of Sciences; University of Chinese Academy of Sciences; Shanghai China

2. School of Public Health; Shanghai Jiao Tong University School of Medicine; China

3. Department of Endocrinology and Metabolism; Zhongshan Hospital; Fudan University; Shanghai China

4. Key Laboratory of Food Safety Risk Assessment; Ministry of Health; Beijing China

Funder

Ministry of Science and Technology of the People's Republic of China

National Natural Science Foundation of China

Chinese Academy of Sciences

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/febs.14294/fullpdf

Reference50 articles.

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4. Evaluation of oxidative stress during apoptosis and necrosis caused by D-galactosamine in rat liver;Sun;Biochem Pharmacol,2003

5. Comprehensive analysis of differential gene expression profiles on D-galactosamine-induced acute mouse liver injury and regeneration;Chung;Toxicology,2006

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