Rosiglitazone via PPAR γ‐dependent suppression of oxidative stress attenuates endothelial dysfunction in rats fed homocysteine thiolactone-Reference-Cited by-同舟云学术

Rosiglitazone via PPAR γ‐dependent suppression of oxidative stress attenuates endothelial dysfunction in rats fed homocysteine thiolactone

Published:2015-02-05 Issue:4 Volume:19 Page:826-835
ISSN:1582-1838
Container-title:Journal of Cellular and Molecular Medicine
language:en
Short-container-title:J. Cell. Mol. Med.

Author:

Yang Xu‐Hong¹,Li Peng¹²,Yin Ya‐Ling¹³,Tu Jiang‐Hua¹,Dai Wen¹,Liu Li‐Ying¹²,Wang Shuang‐Xi⁴

Affiliation:

1. Department of Pharmacology Pharmaceutical College Central South University Changsha China

2. College of Pharmacy Xinxiang Medical University Xinxiang China

3. School of Basic Medical Sciences Xinxiang Medical University Xinxiang China

4. The Key Laboratory of Cardiovascular Remodeling and Function Research Chinese Ministry of Education and Chinese Ministry of Health Qilu Hospital School of Medicine Shandong University Jinan China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.12510

Reference37 articles.

1. Vascular pathology of homocysteinemia: implications for the pathogenesis of arteriosclerosis;McCully KS;Am J Pathol,1969

2. Homocysteine and its thiolactone impair plasmin activity induced by urokinase or streptokinase in vitro

3. The effect of homocysteine-lowering therapy with folic acid on flow-mediated vasodilation in patients with coronary artery disease: A meta-analysis of randomized controlled trials

4. α1D-adrenoceptor-induced relaxation on rat carotid artery is impaired during the endothelial dysfunction evoked in the early stages of hyperhomocysteinemia

5. Roles of PPARs in health and disease

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