Seizure underreporting in LGI1 and CASPR2 antibody encephalitis
Author:
Affiliation:
1. Department of Epileptology University Hospital Bonn, Member of the ERN EpiCARE Bonn Germany
2. Section for Translational Epilepsy Research, Department of Neuropathology University Hospital Bonn, Member of the ERN EpiCARE Bonn Germany
Publisher
Wiley
Subject
Neurology (clinical),Neurology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/epi.17338
Reference18 articles.
1. Antibodies to Kv1 potassium channel-complex proteins leucine-rich, glioma inactivated 1 protein and contactin-associated protein-2 in limbic encephalitis, Morvan’s syndrome and acquired neuromyotonia
2. From VGKC to LGI1 and Caspr2 encephalitis: The evolution of a disease entity over time
3. Autoimmune encephalitis with anti-leucine-rich glioma-inactivated 1 or anti-contactin-associated protein-like 2 antibodies (formerly called voltage-gated potassium channel-complex antibodies)
4. Expanded phenotypes and outcomes among 256 LGI1/CASPR2-IgG-positive patients
5. Evaluation of seizure treatment in anti-LGI1, anti-NMDAR, and anti-GABABR encephalitis
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1. CASPR2‐related epilepsy: A distinctive and unrecognized form of epilepsy in adult and elderly males;Epileptic Disorders;2024-08
2. Risk of Seizure Recurrence Due to Autoimmune Encephalitis With NMDAR, LGI1, CASPR2, and GABA B R Antibodies;Neurology Neuroimmunology & Neuroinflammation;2024-07
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