ASK 1 inhibitor treatment suppresses p38/ JNK signalling with reduced kidney inflammation and fibrosis in rat crescentic glomerulonephritis
Author:
Affiliation:
1. Department of Nephrology Monash Medical Centre Clayton Vic. 3168 Australia
2. Monash University Centre for Inflammatory Diseases Monash Medical Centre Clayton Vic. 3168 Australia
3. Gilead Sciences Inc. Foster City CA USA
Funder
National Health and Medical Research Council
Publisher
Wiley
Subject
Cell Biology,Molecular Medicine
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/jcmm.13705
Reference44 articles.
1. The role of stress-activated protein kinase signaling in renal pathophysiology
2. Compromised MAPK signaling in human diseases: an update
3. The regulatory and signaling mechanisms of the ASK family
4. Abnormal p38 mitogen-activated protein kinase signalling in human and experimental diabetic nephropathy
5. Glomerular and tubular induction of the transcription factor c-Jun in human renal disease
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