Linagliptin‐associated bullous pemphigoid that was most likely caused by IgG autoantibodies against the midportion of BP 180
Author:
Affiliation:
1. Division of Dermatology Niigata University Graduate School of Medical and Dental Sciences Niigata Japan
2. Department of Dermatology Hokkaido University Graduate School of Medicine Sapporo Japan
Publisher
Wiley
Subject
Dermatology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1111/bjd.15111
Reference7 articles.
1. Update on the pathogenesis of bullous pemphigoid: An autoantibody-mediated blistering disease targeting collagen XVII
2. Drug-induced pemphigoid: a review of the literature
3. Autoantibody Profile Differentiates between Inflammatory and Noninflammatory Bullous Pemphigoid
4. Transmembrane collagen XVII, an epithelial adhesion protein, is shed from the cell surface by ADAMs
5. Incretin therapies: highlighting common features and differences in the modes of action of glucagon‐like peptide‐1 receptor agonists and dipeptidyl peptidase‐4 inhibitors
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