Peripheral prion disease pathogenesis is unaltered in the absence of sialoadhesin (Siglec-1/CD169)
Author:
Affiliation:
1. The Roslin Institute and R(D)SVS; University of Edinburgh; Midlothian UK
2. College of Life Sciences; University of Dundee; Dundee UK
Funder
Biotechnology and Biological Sciences Research Council
Publisher
Wiley
Subject
Immunology,Immunology and Allergy
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1111/imm.12294/fullpdf
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