Anti‐β c mAb CSL311 inhibits human nasal polyp pathophysiology in a humanized mouse xenograft model-Reference-Cited by-同舟云学术

Anti‐β c mAb CSL311 inhibits human nasal polyp pathophysiology in a humanized mouse xenograft model

Published:2019-10-03 Issue:2 Volume:75 Page:475-478
ISSN:0105-4538
Container-title:Allergy
language:en
Short-container-title:Allergy

Author:

Yip Kwok Ho¹,Wilson Nicholas J.²,Pant Harshita¹³,Brown Christopher L.⁴,Busfield Samantha²,Ng Milica²,Alhamdoosh Monther²,Woodman Naomi²,Schembri Mark⁵,Tumes Damon J.¹,Vairo Gino²,Lopez Angel F.¹,Nash Andrew D.²,Wilson Michael J.²,Grimbaldeston Michele A.¹,Owczarek Catherine M.²^ORCID

Affiliation:

1. Centre for Cancer Biology SA Pathology and the University of South Australia UniSA CRI Adelaide SA Australia

2. Research and Development CSL Limited Bio21 Institute Parkville Vic. Australia

3. Department of Otolaryngology, Head and Neck Surgery University of Adelaide Adelaide SA Australia

4. Rhinology Clinic Royal Victorian Eye and Ear Hospital East Melbourne Vic. Australia

5. Department of Otolaryngology and Head & Neck Surgery Women's and Children's Hospital Adelaide SA Australia

Funder

Royal Adelaide Hospital

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/all.14041

Reference11 articles.

1. The role of cytokines in chronic rhinosinusitis with nasal polyps

2. Essential mechanisms of differential activation of eosinophils by IL‐3 compared to GM‐CSF and IL‐5;Esnault S;Crit Rev Immunol,2016

3. Signalling by the βc family of cytokines

4. CSL311, a novel, potent, therapeutic monoclonal antibody for the treatment of diseases mediated by the common β chain of the IL-3, GM-CSF and IL-5 receptors

5. Human Nasal Polyp Microenvironments Maintained in a Viable and Functional State as Xenografts in NOD-scid IL2rγnull Mice

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