Association between interleukin‐1 receptor antagonist (IL‐1ra) VNTR, gene polymorphism and breast cancer susceptibility in Iranian population: Experimental and web‐based analysis-Reference-Cited by-同舟云学术

Association between interleukin‐1 receptor antagonist (IL‐1ra) VNTR, gene polymorphism and breast cancer susceptibility in Iranian population: Experimental and web‐based analysis

Published:2022-07-15 Issue:4 Volume:49 Page:254-259
ISSN:1744-3121
Container-title:International Journal of Immunogenetics
language:en
Short-container-title:Int J Immunogenetics

Author:

Ibrahimi Reza¹²,Ibrahimi Mostafa³,jamalzei Behzad³,Akbari Mohammad Esmaeil¹,Navari Mohsen⁴⁵,Moossavi Maryam⁶⁷,Khorasani Milad⁸^ORCID

Affiliation:

1. Cancer Research Center Shahid Beheshti University of Medical Sciences Tehran Iran

2. Student Research Committee Mashhad University of Medical Sciences Mashhad Iran

3. Faculty of Medical Sciences Department of Clinical Biochemistry, Tarbiat Modares University Tehran Iran

4. Department of Laboratory Sciences, School of Paramedical Sciences Torbat Heydariyeh University of Medical Sciences Torbat Heydariyeh Iran

5. Research Center of Advanced Technologies in Medicine Torbat Heydariyeh University of Medical Sciences Torbat Heydariyeh Iran

6. Student Research Committee Birjand University of Medical Sciences Birjand Iran

7. Department of Molecular Medicine Birjand University of Medical Sciences Birjand Iran

8. Department of Biochemistry and Nutrition, School of Medicine Gonabad University of Medical Sciences Gonabad Iran

Publisher

Wiley

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine,Immunology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1111/iji.12584

Reference45 articles.

1. Estradiol, Tamoxifen, and Flaxseed Alter IL-1β and IL-1Ra Levels in Normal Human Breast Tissue in Vivo

2. The Association of IL-1 and HRAS Gene Polymorphisms with Breast Cancer Susceptibility in a Jordanian Population of Arab Descent: A Genotype–Phenotype Study

3. Effects of micro-environment- and malignant cell-derived interleukin-1 in carcinogenesis, tumour invasiveness and tumour–host interactions

4. Inflammation and cancer: back to Virchow?

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