Helicobacter pyloriInduces Serine Phosphorylation of EGFR via Novel TAK1-p38 Activation Pathway in an HB-EGF-Independent Manner-Reference-Cited by-同舟云学术

Helicobacter pyloriInduces Serine Phosphorylation of EGFR via Novel TAK1-p38 Activation Pathway in an HB-EGF-Independent Manner

Published:2015-02-23 Issue:5 Volume:20 Page:381-389
ISSN:1083-4389
Container-title:Helicobacter
language:en
Short-container-title:Helicobacter

Author:

Zaidi Syed Faisal¹²³,Refaat Alaa⁴⁵,Zhou Yue⁶,Sualeh Muhammad Jibran¹,Shin Myoung-Sook⁴,Saiki Ikuo⁴,Sakurai Hiroaki⁶,Sugiyama Toshiro¹

Affiliation:

1. Department of Gastroenterology and Hematology; Graduate School of Medicine and Pharmaceutical Sciences; University of Toyama; Toyama Japan

2. Department of Basic Medical Sciences; College of Medicine; King Saud bin Abdulaziz University of Health Sciences; Jeddah Saudi Arabia

3. Department of Biological and Biomedical Sciences; Faculty of Health Sciences; Aga Khan University; Karachi Pakistan

4. Division of Pathogenic Biochemistry; Institute of Natural Medicine; University of Toyama; Toyama Japan

5. Center for Aging and Associated Diseases; Zewail City of Science and Technology; Giza Egypt

6. Department of Cancer Cell Biology; Graduate School of Medicine and Pharmaceutical Science; University of Toyama; Toyama Japan

Funder

Ministry of Education, Culture, Sports, Science and Technology, Japan

Publisher

Wiley

Subject

Infectious Diseases,Gastroenterology,General Medicine

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1111/hel.12215/fullpdf

Reference40 articles.

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4. The vacuolating cytotoxin of Helicobacter pylori;Cover;Mol Microbiol,1996

5. Helicobacter pylori strain-specific modulation of gastric mucosal cellular turnover: implications for carcinogenesis;Peek;J Gastroenterol,2002

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